myocardial hypertrophy

Diagnostic Value of Multi Slice Spiral Computed Tomography in the Evaluation of Coarctation of Aorta and Interruption Aortic Arch PROTEOMIC ANALYSIS OF MYOCARDIAL HYPERTROPHY INDUCED BY LEFT KIDNEY ARTERY COARCTATION IN RATS

多层螺旋CT在先天性主动脉缩窄和主动脉弓离断诊断中的应用左肾动脉缩窄诱导心肌肥大大鼠心肌蛋白差异表达研究

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PKC is associated closely with multiplication, differentiation, apoptosis of the cells, myocardial hypertrophy and ventricular remodeling.

与细胞增殖 、 分化 、 凋亡 、 心肌肥大、心室重塑等有着密切联系.

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Objective: To investigate the protective effects of total saponins of panax notoginseng ( PNS) on myocardial hypertrophy induced by isoproterenol ( ISO) in rats.

目的：研究三七总皂苷（total saponins of panax notoginseng，PNS）对异丙肾上腺素所致大鼠心肌肥厚的保护作用。方法：用异丙肾上腺素（isoproterenol，ISO）5mg。

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However, PER 2 protein expression of SHR was significantly higher than that of normal rats. CONCLUSION: There might be a positive relationship between hypertension-induced myocardial hypertrophy and aortal disorder and the enhancement of expression of clock protein.

结论：高血压大鼠肥厚心肌和主动脉组织中PER2蛋白表达与正常大鼠中的表达存在差异，高血压导致心肌肥厚和主动脉病变与时钟蛋白表达异常之间可能有着互相促进的关系。

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Objective To explore the effect of Puerarin ( Pue) on cardiac shape and function in myocardial hypertrophy induced by isoproterenol ( Iso).

目的探讨葛根素（Puerarin，Pue）对异丙肾上腺素（Isoproterenol，Iso）所致大鼠心肌肥厚的心脏形态和功能的影响。

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PKG activation inhibits platelet aggregation and myocardial hypertrophy.

PKG活化以后还具有抑制血小板聚集，抑制心肌细胞肥大等功能。

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AIM : To investigate the role of neuropeptide Y ( NPY ) in hypertensive myocardial hypertrophy in rats.

目的: 研究神经肽Y ( NPY ) 在高血压心肌细胞肥大中的作用.

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Conclusion Bre can alleviate myocardial hypertrophy and fibrosis induced by Iso in rats.

结论Bre对Iso引起大鼠心肌肥厚和纤维化具有一定的改善作用.

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They can also relieve the myocardial hypertrophy and improve the cardionephric function.

同时还具有抑制心肌肥厚,改善心、肾功能等作用.

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Conclusion Long-term treatment with taurine can inhibit cardiomyocyte apoptosis and reverse myocardial hypertrophy, and both effects are related to its inhibition of tissue renin-angiotensin system ( RAS) in renovascular hypertensive rats.

结论长疗程牛磺酸治疗可抑制高血压大鼠心肌细胞凋亡和逆转心肌肥厚，上述作用与药物拮抗组织RAS有关。

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Conclusion: Captopril may effectively prevent and treat myocardial hypertrophy.

结论: 卡托普利能有效防治心肌肥厚的发生.

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Conclusion In the myocardial hypertrophy models, cardiac ADM increased may due to its local anti-function on myocardial hypertrophy as an autocrine and ( or) paracrine hormone combined with ANP.

结论在心肌肥厚模型中，心肌ADM升高可能是作为心肌自分泌和/或旁分泌激素在局部起抗心肌肥厚的作用，ADM和ANP可能组成抗肥厚的调节组合。

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